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 Wysłany: ¦ro 11:55, 23 Mar 2011 Temat postu: clarks shoes outlet The etiology and pathogenesis |
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The etiology and pathogenesis of peptic ulcer
Secretion decreased, then at the weak of stomach acid in the gastric mucosa and its protection I ③ gastric emptying disorders, digesta remain too long in the antrum, while the bow j excessive stimulation of gastrin secretion from the secretion of gastric acid ; ④ stomach and {. B: refers to the intestinal spasm, reducing blood supply Zhu open by the profile surface is not conducive to the more mucosal injury, such as Taiwan. Which is a contradiction between gastric acid and pepsin leaving damage as a major area into an important factor, particularly important for the majority of the role of gastric acid in patients with duodenal ulcer gastric acid secretion of wax added boat, apparently consistent with this mechanism. Gastric ulcer patients with normal or lower than the cocoon, but the feeling in the stomach, revolves around the destruction of barriers, based on the penetration of hydrogen ions back into the main damage factor in ulcer formation, it is still an important role in gastric acid. Who proved not suffering from peptic ulcer without stomach acid. The incidence of gastric ulcer protective factors focused on the weakening performance of the gastric mucosal barrier breakdown, loss of motor function pyloric withered gastritis and duodenal flow and buildings reflect the anti-stick injury to the stomach diminished capacity and so on. Normally, gastric mucosa from stomach with the total property damage to passengers or repair an injury shortly after gastric mucosal barrier. Strong gastric epithelial cell membrane of the lipid barrier of white layer is an important part of the election, when the destruction of the hydrogen from the gastric juice to seep back to stick to Cou layer, causing histamine release, serum white ink, blood oozing, increase the secretion of stomach enzymes and the deaf white blood stasis caused by partial damage to gastric mucosa in the short term lead to rotten elk (gastritis) can lead to long term gastric ulcer, who can dissolve the fat based material can damage this layer sets deaf white fat such substances with aspirin, alcohol and other drugs and the body's bile and pancreatic duodenal juice components such disclosure. In gastric ulcer patients, often appear pyloric motor function disorders (pyloric sphincter relaxation), a large inflow of duodenal juice to counter the stomach,clarks shoes outlet, the stomach kwu membrane barrier damage, leading to ulcer formation. In addition, the Ministry of gastrin and gastric rustic retention factor by the gastric antrum of the stomach aldehyde cells (G cells) secreted by the parietal cells of gastrin has excited so if the role of gastric acid secretion. In gastric ulcer patients, the vagus nerve dysfunction often so weak alkaline gastric motility, gastric retention appeared excited G cells, the results of gastrin secretion index, which may eventually cause ulcers. In addition, many village doctors in China, Journal of Composite ulcers, often first, duodenal ulcer, which caused a lot of obstacles to the emergence of Helicobacter pylori gastric emptying of the Ministry of honey left side, and then cause ulcers, which are complex sets ulcer important principle. The incidence of duodenal ulcer damage factors are focused on enhanced performance of the overall increase of parietal cells, neuroendocrine dysfunction caused by gastric acid secretion and so on. Acid secretion of parietal cells are cells called parietal cells in the total amount of the total. Duodenal ulcer patients more than the total number of parietal cells increased above the normal population J l times, while the parietal cells of gastric ulcer patients were normal or slightly smaller than the number of normal. Therefore, the total number of parietal cells increased the occurrence of a close relationship with duodenal ulcer. Nervous system and endocrine, the cerebral cortex and hypothalamus in the following two ways regulate gastrointestinal secretion was, digestion, movement and other functions and blood circulation. ① autonomic dish pit (before the hypothalamic nuclei in vagus nerve vagus nerve); ② endocrine system (hypothalamus recite one kidney on a vertical gap axis). Abnormal vagus nerve Fen Wang is too high to make gastric acid in the pathogenesis of duodenal ulcer play an important role. Autonomic nervous system is well received by the regulation of the cerebral cortex, the brain dysfunction of the skin is often the flag above the vagus nerve causes abnormal increase of excitability. Understandable from the spirit of continued and excessive stress, neuropsychiatric conditions such as brand-emotional factors in health and recurrence of duodenal ulcer divinatory share of prominence. Overall the incidence of peptic ulcer are mostly interrelated factors or a combination, constitute a series of links in the pathogenesis of, and different patients often have their different risk factors. Therefore,GHD glätteisen, we must end in the clinical history and examination data sets to explore fully the pathogenesis of each case sets of factors in order to brake for better defense at the Fu. Clinical manifestations and diagnosis of peptic ulcer thinking Medical seeking a white hospital bed startled expression Chi Baorong chronic process, ranging from a few years,herve leger sale, as many as lO years, even decades, a chronic, disease onset and remission often seen Alternating. Attacks and more of the following factors: season (winter and late fall), mental stress, mood swings, diet adjustment, drugs. Reflect a non-ulcer activity in remission or from Taichung} rhythmic pain, ulcers and more in the postprandial 1 / 2 tame,Christian Louboutin Ireland, disappear when the next meal, duodenal ulcer 3 to 4 hours after meal there , continuing until the next meal, eating remission, also known as fasting pain. Or at night before going to bed at night pain, pain, said. Prepyloric ulcer area similar to the Rhythm and duodenal ulcer, pyloric ulcer often without rhythm. Complications disappear when the cyclical and rhythmic, and other gastrointestinal symptoms, returned to acid, belching, nausea,
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