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 Wysłany: Czw 20:14, 10 Mar 2011 Temat postu: tory burch reva kdk xbf aewb duk |
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,tory burch reva
P53 protein overexpression in laryngeal cancer
Protein half-life extended. Nucleus in the concentration of small packets can be achieved immunohistochemistry findings. Mutation P; s protein has lost the normal tumor suppressor function '. Our experiments confirmed the fine roar of laryngeal cancer P protein overexpression, fluorescence staining was 68.0 (17/25), in line with foreign literature, suggesting that laryngeal cancer gene mutations present in Pj,UGG stivali, and its expression of the protein lost normal function of the nuclear accumulation of small roar,mbt shoes utlet, can not regulate the normal growth of small roar. Uncontrolled proliferation of malignant cells to produce. This is in the development of laryngeal carcinoma is an important mechanism. As to whether the role of alien virus, which causes the Psa gene mutation. Further study,GHD glätteisen, pre ~ rgene. Nature. 199l: 35l: 2MaestroR. DolcettiRGaspar ~ ttoD. eta1. FIighfriquen-cyofP3alterationsassociatedwithproteinoverexpressioninhumansqual ~) ous-cellcarcinomaofthelarynxOn ~ ogene · I992: 7:1 _59.3AnwarK · NakakukiK'ImaiH'etOverexpression. fPproteininhuman] aryngealcarcinoma. 1ntJCan-eer. 1993; 03:9524 ChengJ. HaasM. FrequentmutationsinthePcumorsuppressorgeneinhumanleukemiaT-ce1] lines. MolCeLl both ol, l990 {10:55025 GannonJV. LaneDP. 3andDNApo] ymeraseacorn-peteforbindingtoSVi. Tantigen. Nature. 1987:329:4500 Werne ~ BA. LevineAJ, How [eyPM. Associationofhu-manpapillomavirustype16andI8EproteinswithPScience. I crucian 0 {248:706 (Received 1994-08 a O2,mulberry bags, refurbished I994-09-10) |
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