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 Wysłany: Czw 20:14, 10 Mar 2011 Temat postu: tory burch reva kdk xbf aewb duk |
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,[link widoczny dla zalogowanych]
P53 protein overexpression in laryngeal cancer
Protein half-life extended. Nucleus in the concentration of small packets can be achieved immunohistochemistry findings. Mutation P; s protein has lost the normal tumor suppressor function '. Our experiments confirmed the fine roar of laryngeal cancer P protein overexpression, fluorescence staining was 68.0 (17/25), in line with foreign literature, suggesting that laryngeal cancer gene mutations present in Pj,[link widoczny dla zalogowanych], and its expression of the protein lost normal function of the nuclear accumulation of small roar,[link widoczny dla zalogowanych], can not regulate the normal growth of small roar. Uncontrolled proliferation of malignant cells to produce. This is in the development of laryngeal carcinoma is an important mechanism. As to whether the role of alien virus, which causes the Psa gene mutation. Further study,[link widoczny dla zalogowanych], pre ~ rgene. Nature. 199l: 35l: 2MaestroR. DolcettiRGaspar ~ ttoD. eta1. FIighfriquen-cyofP3alterationsassociatedwithproteinoverexpressioninhumansqual ~) ous-cellcarcinomaofthelarynxOn ~ ogene · I992: 7:1 _59.3AnwarK · NakakukiK'ImaiH'etOverexpression. fPproteininhuman] aryngealcarcinoma. 1ntJCan-eer. 1993; 03:9524 ChengJ. HaasM. FrequentmutationsinthePcumorsuppressorgeneinhumanleukemiaT-ce1] lines. MolCeLl both ol, l990 {10:55025 GannonJV. LaneDP. 3andDNApo] ymeraseacorn-peteforbindingtoSVi. Tantigen. Nature. 1987:329:4500 Werne ~ BA. LevineAJ, How [eyPM. Associationofhu-manpapillomavirustype16andI8EproteinswithPScience. I crucian 0 {248:706 (Received 1994-08 a O2,[link widoczny dla zalogowanych], refurbished I994-09-10)
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